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rdf:type |
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lifeskim:mentions |
umls-concept:C0030705,
umls-concept:C0085413,
umls-concept:C0162867,
umls-concept:C0205042,
umls-concept:C0221099,
umls-concept:C0439830,
umls-concept:C0741968,
umls-concept:C0806140,
umls-concept:C1256463,
umls-concept:C1280412,
umls-concept:C1522318,
umls-concept:C2347957
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pubmed:issue |
4
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pubmed:dateCreated |
2008-6-30
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pubmed:abstractText |
Cardiovascular problems are a major cause of morbidity and mortality in patients with autosomal dominant polycystic kidney disease. Endothelial dysfunction, an early and reversible feature in the pathogenesis of atherosclerosis, is associated with increased vascular smooth muscle tone, arterial stiffening, and increased intima-media thickness. Coronary flow velocity reserve is a noninvasive test showing endothelial function of epicardial coronary arteries and coronary microcirculatory function. The aim of the study was to investigate the carotid intima-media thickness and coronary flow velocity reserve in patients with autosomal dominant polycystic kidney disease.
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-10232688,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-10704159,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-10779454,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-10878400,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-11134267,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-11790699,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-12171785,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-12372574,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-1244564,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-12631134,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-12968086,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-12969157,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-138494,
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http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-15264179,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-15542287,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-15792499,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-16221225,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-17493464,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-17917317,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-1838571,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-2193495,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-2215576,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-6846334,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-709763,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-7397962,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-7579053,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-7819937,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-7908078,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-8321262,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-8490116,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-9259356,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-9362404,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-9593560,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-9825400,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18354076-9856512
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
1555-905X
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pubmed:author |
pubmed-author:AlisirSabahatS,
pubmed-author:CimenArif OAO,
pubmed-author:EcderTevfikT,
pubmed-author:ElitokAliA,
pubmed-author:KasikciogluErdemE,
pubmed-author:NamliSuleS,
pubmed-author:OflazHuseyinH,
pubmed-author:TufanFatihF,
pubmed-author:TurkmenKultiginK,
pubmed-author:UsluBoraB,
pubmed-author:UysalMukreminM
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pubmed:issnType |
Electronic
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pubmed:volume |
3
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
986-91
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pubmed:dateRevised |
2009-11-18
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pubmed:meshHeading |
pubmed-meshheading:18354076-Adult,
pubmed-meshheading:18354076-Atherosclerosis,
pubmed-meshheading:18354076-Blood Flow Velocity,
pubmed-meshheading:18354076-Carotid Arteries,
pubmed-meshheading:18354076-Case-Control Studies,
pubmed-meshheading:18354076-Coronary Circulation,
pubmed-meshheading:18354076-Coronary Vessels,
pubmed-meshheading:18354076-Echocardiography, Stress,
pubmed-meshheading:18354076-Female,
pubmed-meshheading:18354076-Humans,
pubmed-meshheading:18354076-Kidney Tubules,
pubmed-meshheading:18354076-Male,
pubmed-meshheading:18354076-Middle Aged,
pubmed-meshheading:18354076-Polycystic Kidney, Autosomal Dominant,
pubmed-meshheading:18354076-Tunica Intima,
pubmed-meshheading:18354076-Tunica Media
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pubmed:year |
2008
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pubmed:articleTitle |
Coronary flow velocity reserve and carotid intima media thickness in patients with autosomal dominant polycystic kidney disease: from impaired tubules to impaired carotid and coronary arteries.
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pubmed:affiliation |
Department of Internal Medicine, Istanbul School of Medicine, Istanbul, Turkey.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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