18309114

Source:http://linkedlifedata.com/resource/pubmed/id/18309114

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0011209, umls-concept:C0205178, umls-concept:C0205276, umls-concept:C0916207, umls-concept:C1261287, umls-concept:C1267092, umls-concept:C1292733, umls-concept:C1416611, umls-concept:C1522318, umls-concept:C1549649, umls-concept:C1709059, umls-concept:C2349209
pubmed:issue	6
pubmed:dateCreated	2008-5-22
pubmed:abstractText	We previously demonstrated that upregulation of intermediate-conductance Ca(2+)-activated K(+) channels (K(Ca)3.1) is necessary for mitogen-induced phenotypic modulation in isolated porcine coronary smooth muscle cells (SMCs). The objective of the present study was to determine the role of K(Ca)3.1 in the regulation of coronary SMC phenotypic modulation in vivo using a swine model of postangioplasty restenosis.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/HL 52490, http://linkedlifedata.com/resource/pubmed/grant/P01 HL052490-130006
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pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9505803
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Intermediate-Conductance..., http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channel Blockers, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins c-fos, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins c-jun, http://linkedlifedata.com/resource/pubmed/chemical/Pyrazoles, http://linkedlifedata.com/resource/pubmed/chemical/TRAM 34, http://linkedlifedata.com/resource/pubmed/chemical/Transcription Factors
pubmed:status	MEDLINE
pubmed:month	Jun
pubmed:issn	1524-4636
pubmed:author	pubmed-author:BowlesD KDK, pubmed-author:CheongAA, pubmed-author:RamanGG, pubmed-author:TharpD LDL, pubmed-author:WamhoffB RBR, pubmed-author:WulffHH
pubmed:issnType	Electronic
pubmed:volume	28
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	1084-9
pubmed:dateRevised	2010-11-18
pubmed:meshHeading	pubmed-meshheading:18309114-Angioplasty, Balloon, Coronary, pubmed-meshheading:18309114-Animals, pubmed-meshheading:18309114-Cell Differentiation, pubmed-meshheading:18309114-Coronary Stenosis, pubmed-meshheading:18309114-Coronary Vessels, pubmed-meshheading:18309114-Disease Models, Animal, pubmed-meshheading:18309114-Intermediate-Conductance Calcium-Activated Potassium..., pubmed-meshheading:18309114-Male, pubmed-meshheading:18309114-Muscle, Smooth, Vascular, pubmed-meshheading:18309114-Phenotype, pubmed-meshheading:18309114-Potassium Channel Blockers, pubmed-meshheading:18309114-Proto-Oncogene Proteins c-fos, pubmed-meshheading:18309114-Proto-Oncogene Proteins c-jun, pubmed-meshheading:18309114-Pyrazoles, pubmed-meshheading:18309114-Swine, pubmed-meshheading:18309114-Transcription Factors
pubmed:year	2008
pubmed:articleTitle	Local delivery of the KCa3.1 blocker, TRAM-34, prevents acute angioplasty-induced coronary smooth muscle phenotypic modulation and limits stenosis.

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