Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1-2
pubmed:dateCreated
2008-4-16
pubmed:abstractText
An Arabidopsis mutant with improved salt tolerant germination was isolated from a T-DNA insertion library and designated as AT6. This mutant also exhibited improved salt tolerance phenotype in later developmental stages. But no apparent difference was observed in response to ABA, GA or ethylene during germination between the mutant and the wildtype. The T-DNA was inserted in the At1g73660 locus that coded for a putative MAPKKK. Genetic and multiple mutant allele analyses confirmed that the knockout of this gene resulted in improved salt tolerance phenotype and provided strong evidence that the genetic locus At1g73660 negatively regulated salt tolerance in Arabidopsis. The At1g73660 was down regulated in response to salt stress in the mutants, which is consistent with its role as a negative regulator. It is therefore hypothesized that the AT1g73660 may serve as one of the off-switches of stress responses that are required for unstressed conditions.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0167-4412
pubmed:author
pubmed:issnType
Print
pubmed:volume
67
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
125-34
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed:year
2008
pubmed:articleTitle
The genetic locus At1g73660 encodes a putative MAPKKK and negatively regulates salt tolerance in Arabidopsis.
pubmed:affiliation
School of Life Sciences, University of Science and Technology of China, Hefei, Anhui, China.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't