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| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C0021853 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C0077503 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C0026565 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:18242213 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:18242213 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:18242213 | pubmed:dateCreated | 2008-2-4 | lld:pubmed |
| pubmed-article:18242213 | pubmed:abstractText | Ligation of tumor necrosis factor (TNF) receptors (TNFRs) with TNF plays a critical role in the pathogenesis of human inflammatory bowel disease (IBD). However, it remains unclear which cell types activated through TNFR-associated signaling cascades are involved in the pathogenesis of colitis. | lld:pubmed |
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| pubmed-article:18242213 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18242213 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18242213 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18242213 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:18242213 | pubmed:issn | 1528-0012 | lld:pubmed |
| pubmed-article:18242213 | pubmed:author | pubmed-author:MizoguchiEmik... | lld:pubmed |
| pubmed-article:18242213 | pubmed:author | pubmed-author:MizoguchiAtsu... | lld:pubmed |
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| pubmed-article:18242213 | pubmed:author | pubmed-author:SugimotoKenK | lld:pubmed |
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| pubmed-article:18242213 | pubmed:author | pubmed-author:NagataniKatsu... | lld:pubmed |
| pubmed-article:18242213 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18242213 | pubmed:volume | 134 | lld:pubmed |
| pubmed-article:18242213 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18242213 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18242213 | pubmed:pagination | 470-80 | lld:pubmed |
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| pubmed-article:18242213 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18242213 | pubmed:articleTitle | TNF receptor type I-dependent activation of innate responses to reduce intestinal damage-associated mortality. | lld:pubmed |
| pubmed-article:18242213 | pubmed:affiliation | Center for the Study of Inflammatory Bowel Disease, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA. emizoguchi@partners.org | lld:pubmed |
| pubmed-article:18242213 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18242213 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18242213 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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