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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2008-2-4
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pubmed:abstractText |
Ligation of tumor necrosis factor (TNF) receptors (TNFRs) with TNF plays a critical role in the pathogenesis of human inflammatory bowel disease (IBD). However, it remains unclear which cell types activated through TNFR-associated signaling cascades are involved in the pathogenesis of colitis.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
1528-0012
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
134
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
470-80
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pubmed:meshHeading |
pubmed-meshheading:18242213-Animals,
pubmed-meshheading:18242213-Apoptosis,
pubmed-meshheading:18242213-Bone Marrow,
pubmed-meshheading:18242213-Bone Marrow Transplantation,
pubmed-meshheading:18242213-Cell Proliferation,
pubmed-meshheading:18242213-Colitis,
pubmed-meshheading:18242213-Colon,
pubmed-meshheading:18242213-Dextran Sulfate,
pubmed-meshheading:18242213-Disease Models, Animal,
pubmed-meshheading:18242213-Homeodomain Proteins,
pubmed-meshheading:18242213-Homeostasis,
pubmed-meshheading:18242213-Intestinal Mucosa,
pubmed-meshheading:18242213-Mice,
pubmed-meshheading:18242213-Mice, Inbred C57BL,
pubmed-meshheading:18242213-Mice, Knockout,
pubmed-meshheading:18242213-Receptors, Tumor Necrosis Factor, Type I,
pubmed-meshheading:18242213-Receptors, Tumor Necrosis Factor, Type II,
pubmed-meshheading:18242213-Severity of Illness Index,
pubmed-meshheading:18242213-Signal Transduction
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pubmed:year |
2008
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pubmed:articleTitle |
TNF receptor type I-dependent activation of innate responses to reduce intestinal damage-associated mortality.
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pubmed:affiliation |
Center for the Study of Inflammatory Bowel Disease, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA. emizoguchi@partners.org
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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