Linked Life Data

1821755

Source:http://linkedlifedata.com/resource/pubmed/id/1821755

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
1992-8-27
pubmed:abstractText
A brief description of the fundamental concepts of the biology and pathophysiology of excitatory amino acids, lactic acidosis, and free radicals in brain injury have been presented. For a comprehensive review of these topics, the references provide access to an extensive body of research in each of these areas. They represent a complex series of interrelated events that are set into motion at the initial injury. Some neurons are destined to die despite all therapeutic endeavors, but further investigation into the mechanisms responsible for delayed neuronal demise are in the early stages of human investigation or very close to clinical trials. Efforts directed at providing a favorable extracellular milieu for those neurons that are salvageable provide promise for continued improvements in outcome for patients with head injury.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
1042-3680
pubmed:author
pubmed:issnType
Print
pubmed:volume
2
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
483-91
pubmed:dateRevised
2005-11-16
pubmed:meshHeading
pubmed:year
1991
pubmed:articleTitle
Changes in the treatment of head injury.
pubmed:affiliation
Department of Neurosurgery, University of California, School of Medicine, Los Angeles.
pubmed:publicationType
Journal Article, Review