GENERIC 18193548

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0037083, umls-concept:C0441712, umls-concept:C1710082, umls-concept:C2610891
pubmed:issue	1
pubmed:dateCreated	2008-1-11
pubmed:abstractText	Glucocorticoid-induced T-cell apoptosis is supposed to require the change of gene expression. Enhanced expression of proapoptotic gene or inhibited expression of antiapoptotic gene seems to be necessary, although the real target gene remain to be elucidated. Recently, it was reported that extracellular signal-regulated protein kinase (ERK) inhibits glucocorticoid-induced T-cell apoptosis. This finding might provide new tools for overcoming glucocorticoid-resistance.
pubmed:language	jpn
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0420546
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Glucocorticoids
pubmed:status	MEDLINE
pubmed:month	Jan
pubmed:issn	0047-1852
pubmed:author	pubmed-author:OkabeTaijiroT, pubmed-author:TakayanagiRyouichiR, pubmed-author:YanaseToshihikoT
pubmed:issnType	Print
pubmed:volume	66
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	94-7
pubmed:dateRevised	2011-7-27
pubmed:meshHeading	pubmed-meshheading:18193548-Animals, pubmed-meshheading:18193548-Apoptosis, pubmed-meshheading:18193548-Glucocorticoids, pubmed-meshheading:18193548-Mice, pubmed-meshheading:18193548-T-Lymphocytes
pubmed:year	2008
pubmed:articleTitle	[The signaling mechanism of glucocorticoid-induced T-cell apoptosis].
pubmed:affiliation	Department of Medicine and Bioregulatory Science, Graduate School of Medical Science, Kyushu University.
pubmed:publicationType	Journal Article, English Abstract, Review