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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2008-5-15
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pubmed:abstractText |
Perihemorrhagic pathophysiology of spontaneous intracerebral hemorrhages (ICH) remains unclear. Recently, ischemic changes in the perihemorrhagic zone (PHZ) have been discussed as a potential source of secondary damage. In this study, we focussed on diffusion and perfusion characteristics of experimental ICH.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:issn |
1541-6933
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
8
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
448-55
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pubmed:meshHeading |
pubmed-meshheading:18188706-Animals,
pubmed-meshheading:18188706-Brain,
pubmed-meshheading:18188706-Brain Edema,
pubmed-meshheading:18188706-Brain Ischemia,
pubmed-meshheading:18188706-Cerebral Hemorrhage,
pubmed-meshheading:18188706-Cerebrovascular Circulation,
pubmed-meshheading:18188706-Disease Models, Animal,
pubmed-meshheading:18188706-Hematoma,
pubmed-meshheading:18188706-Magnetic Resonance Imaging,
pubmed-meshheading:18188706-Male,
pubmed-meshheading:18188706-Rats,
pubmed-meshheading:18188706-Rats, Wistar
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pubmed:year |
2008
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pubmed:articleTitle |
MRI of the perihemorrhagic zone in a rat ICH model: effect of hematoma evacuation.
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pubmed:affiliation |
Department of Neurosurgery, University Hospital Heidelberg, Im Neuenheimer Feld 400, Heidelberg, Germany. berk.orakcioglu@med.uni-heidelberg.de
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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