Linked Life Data

18097150

Source:http://linkedlifedata.com/resource/pubmed/id/18097150

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2008-2-13
pubmed:abstractText
Apoptosis has been implicated in renal ischemic injury, the regulating mechanism of which is still unclear. Signal transducers and activators of transcription (STAT) participate in inflammation, apoptosis, and tumorigenesis. In the in vitro model of renal ischemic injury, we explored the role of the STAT3, a major component of the STAT family, in apoptosis of human proximal tubular epithelial cell (HKC) induced by ATP depletion/recovery.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1660-2129
pubmed:author
pubmed:copyrightInfo
(c) 2007 S. Karger AG, Basel
pubmed:issnType
Electronic
pubmed:volume
108
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
e11-8
pubmed:meshHeading
pubmed:year
2008
pubmed:articleTitle
STAT3 inhibits apoptosis of human renal tubular epithelial cells induced by ATP depletion/recovery.
pubmed:affiliation
Department of Nephrology, General Hospital of PLA, Nephrology Institute and Key Laboratory of PLA, Beijing, PR China.
pubmed:publicationType
Journal Article, Comparative Study, Research Support, Non-U.S. Gov't