Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2008-3-17
pubmed:abstractText
Atherosclerosis is the leading cause of death in Western societies and a chronic inflammatory disease. However, the key mediators linking recruitment of inflammatory cells to atherogenesis remain poorly defined. Poly(ADP-ribose) polymerase 1 (PARP1) is a nuclear enzyme, which plays a role in acute inflammatory diseases.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Apolipoproteins E, http://linkedlifedata.com/resource/pubmed/chemical/Cell Adhesion Molecules, http://linkedlifedata.com/resource/pubmed/chemical/Cholesterol, http://linkedlifedata.com/resource/pubmed/chemical/E-Selectin, http://linkedlifedata.com/resource/pubmed/chemical/Enzyme Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/Inflammation Mediators, http://linkedlifedata.com/resource/pubmed/chemical/N-(oxo-5,6-dihydrophenanthridin-2-yl..., http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type II, http://linkedlifedata.com/resource/pubmed/chemical/Nos2 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/P-Selectin, http://linkedlifedata.com/resource/pubmed/chemical/Parp1 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Phenanthrenes, http://linkedlifedata.com/resource/pubmed/chemical/Poly(ADP-ribose) Polymerases, http://linkedlifedata.com/resource/pubmed/chemical/Vascular Cell Adhesion Molecule-1
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
0008-6363
pubmed:author
pubmed:issnType
Print
pubmed:day
1
pubmed:volume
78
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
158-66
pubmed:meshHeading
pubmed-meshheading:18093987-Animals, pubmed-meshheading:18093987-Apolipoproteins E, pubmed-meshheading:18093987-Atherosclerosis, pubmed-meshheading:18093987-Cell Adhesion Molecules, pubmed-meshheading:18093987-Cholesterol, pubmed-meshheading:18093987-Disease Models, Animal, pubmed-meshheading:18093987-E-Selectin, pubmed-meshheading:18093987-Enzyme Inhibitors, pubmed-meshheading:18093987-Inflammation, pubmed-meshheading:18093987-Inflammation Mediators, pubmed-meshheading:18093987-Macrophages, pubmed-meshheading:18093987-Male, pubmed-meshheading:18093987-Mice, pubmed-meshheading:18093987-Mice, Inbred C57BL, pubmed-meshheading:18093987-Mice, Knockout, pubmed-meshheading:18093987-Necrosis, pubmed-meshheading:18093987-Nitric Oxide Synthase Type II, pubmed-meshheading:18093987-P-Selectin, pubmed-meshheading:18093987-Phenanthrenes, pubmed-meshheading:18093987-Poly(ADP-ribose) Polymerases, pubmed-meshheading:18093987-T-Lymphocytes, pubmed-meshheading:18093987-Vascular Cell Adhesion Molecule-1
pubmed:year
2008
pubmed:articleTitle
PARP1 is required for adhesion molecule expression in atherogenesis.
pubmed:affiliation
Cardiovascular Research, Institute of Physiology, University of Zurich and Cardiology, Cardiovascular Center, University Hospital Zurich, Winterthurerstrasse 190, Zurich CH-8057, Switzerland.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't