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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2008-3-3
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pubmed:abstractText |
Pulmonary veins are the most important focus for the generation of atrial fibrillation. Abnormal calcium homeostasis with ryanodine receptor dysfunction may underlie the arrhythmogenic activity in pulmonary veins. The preferential ryanodine receptor stabilizer (K201) possesses antiarrhythmic effects through calcium regulation. The purpose of this study was to investigate the effects of K201 on the arrhythmogenic activity and calcium regulation of pulmonary vein cardiomyocytes.
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-10230854,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-10545432,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-10830164,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-10864882,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-11085966,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-11090108,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-11733406,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-11744028,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-11998974,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-12551874,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-12668519,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-12837242,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-12890053,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-1394921,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15041695,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15073377,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15149427,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15313939,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15621038,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15851156,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15851612,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15922271,
http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-15927973,
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http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-17313373,
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http://linkedlifedata.com/resource/pubmed/commentcorrection/17994112-9725923
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0007-1188
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
153
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
915-25
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pubmed:dateRevised |
2009-11-18
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pubmed:meshHeading |
pubmed-meshheading:17994112-Action Potentials,
pubmed-meshheading:17994112-Animals,
pubmed-meshheading:17994112-Anti-Arrhythmia Agents,
pubmed-meshheading:17994112-Arrhythmias, Cardiac,
pubmed-meshheading:17994112-Calcium,
pubmed-meshheading:17994112-Calcium Channels, L-Type,
pubmed-meshheading:17994112-Dose-Response Relationship, Drug,
pubmed-meshheading:17994112-Homeostasis,
pubmed-meshheading:17994112-Isoproterenol,
pubmed-meshheading:17994112-Myocytes, Cardiac,
pubmed-meshheading:17994112-Pulmonary Veins,
pubmed-meshheading:17994112-Rabbits,
pubmed-meshheading:17994112-Sarcoplasmic Reticulum,
pubmed-meshheading:17994112-Sodium-Calcium Exchanger,
pubmed-meshheading:17994112-Thiazepines
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pubmed:year |
2008
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pubmed:articleTitle |
Effect of K201, a novel antiarrhythmic drug on calcium handling and arrhythmogenic activity of pulmonary vein cardiomyocytes.
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pubmed:affiliation |
Division of Cardiovascular Medicine, Taipei Medical University-Wan Fang Hospital, Taipei, Taiwan. a9900112@,s15.hinet.net
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
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