Linked Life Data

17967972

Source:http://linkedlifedata.com/resource/pubmed/id/17967972

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
20
pubmed:dateCreated
2007-11-13
pubmed:abstractText
CD40 is a costimulatory molecule that acts as a central mediator of various immune responses, including those involved in the progression of atherosclerosis. Correspondent to its function, CD40 is present not only on many immune cells, such as antigen-presenting cells and T cells, but also on nonimmune cells, such as endothelial cells.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
13
pubmed:volume
116
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2288-97
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed-meshheading:17967972-Activin Receptors, Type II, pubmed-meshheading:17967972-Animals, pubmed-meshheading:17967972-Antigens, CD40, pubmed-meshheading:17967972-Aorta, Thoracic, pubmed-meshheading:17967972-Capillaries, pubmed-meshheading:17967972-Coculture Techniques, pubmed-meshheading:17967972-Down-Regulation, pubmed-meshheading:17967972-Endothelial Cells, pubmed-meshheading:17967972-Femoral Artery, pubmed-meshheading:17967972-Gene Expression, pubmed-meshheading:17967972-Humans, pubmed-meshheading:17967972-Immunohistochemistry, pubmed-meshheading:17967972-Mesenteric Arteries, pubmed-meshheading:17967972-Mesenteric Veins, pubmed-meshheading:17967972-Mice, pubmed-meshheading:17967972-Mice, Inbred C57BL, pubmed-meshheading:17967972-Myocytes, Smooth Muscle, pubmed-meshheading:17967972-Stress, Mechanical, pubmed-meshheading:17967972-Transforming Growth Factor beta1, pubmed-meshheading:17967972-Umbilical Veins, pubmed-meshheading:17967972-Up-Regulation
pubmed:year
2007
pubmed:articleTitle
Cyclic stretch controls the expression of CD40 in endothelial cells by changing their transforming growth factor-beta1 response.
pubmed:affiliation
University Hospital Heidelberg, Institute of Physiology and Pathophysiology, Division of Cardiovascular Physiology, Im Neuenheimer Feld 326, 69120 Heidelberg, Germany.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't