Linked Life Data

1795207

Source:http://linkedlifedata.com/resource/pubmed/id/1795207

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
8
pubmed:dateCreated
1992-4-9
pubmed:abstractText
A major problem confronting behavioural theories of hypertension, such as the reactivity hypothesis, is that stress is likely to be intermittent, whereas the early stages of hypertension appear to be characterized by an increase in the tonic level of blood pressure and sympathetic activity. Furthermore, intermittent sympathetic arousal (e.g. exercise, thigh-cuff compression) does not necessarily raise tonic blood pressure. A worksite-based study of occupational stress has indicated that people in high-stress jobs have increased blood pressure throughout the day and night, which is at least consistent with a behaviourally mediated resetting of the tonic blood pressure level. There is evidence that adrenaline is preferentially released in response to behavioural stresses. According to the 'adrenaline hypothesis', adrenaline can raise tonic blood pressure while noradrenaline does not. We therefore propose that the different long-term effects of behavioural stress and exercise on blood pressure can be explained by their differing effects on catecholamine release.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
0952-1178
pubmed:author
pubmed:issnType
Print
pubmed:volume
9
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
S66-8
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed:year
1991
pubmed:articleTitle
Can behavioural factors produce a sustained elevation of blood pressure? Some observations and a hypothesis.
pubmed:affiliation
Cardiovascular Center, New York Hospital-Cornell University Center, New York, NY 10021.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Review