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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2007-12-10
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pubmed:abstractText |
The pathophysiological mechanism in cardiac syndrome X has been suggested as impairment in normal endothelial function of the coronary microvasculature, resulting in inadequate flow reserve. However, despite the extensive studies, the precise mechanisms in cardiac syndrome X remain unclear.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
1096-0023
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pubmed:author |
|
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pubmed:issnType |
Electronic
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pubmed:volume |
40
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
172-6
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pubmed:meshHeading |
pubmed-meshheading:17945505-Adult,
pubmed-meshheading:17945505-Blood Cell Count,
pubmed-meshheading:17945505-C-Reactive Protein,
pubmed-meshheading:17945505-Chronic Disease,
pubmed-meshheading:17945505-Coronary Circulation,
pubmed-meshheading:17945505-Endothelium, Vascular,
pubmed-meshheading:17945505-Female,
pubmed-meshheading:17945505-Humans,
pubmed-meshheading:17945505-Inflammation,
pubmed-meshheading:17945505-Inflammation Mediators,
pubmed-meshheading:17945505-Interleukin-6,
pubmed-meshheading:17945505-Microvascular Angina,
pubmed-meshheading:17945505-Middle Aged,
pubmed-meshheading:17945505-Prospective Studies
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pubmed:year |
2007
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pubmed:articleTitle |
Elevated circulating inflammatory markers in female patients with cardiac syndrome X.
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pubmed:affiliation |
Department of Cardiology, Fu Wai Hospital, Chinese Academy of Medical Sciences, Peking Union Medical College, Beijing 100037, PR China. lijnjn@yahoo.com.cn
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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