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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
6
|
| pubmed:dateCreated |
1992-4-8
|
| pubmed:abstractText |
Type I diabetes occurs as a result of T-cell-mediated beta-cell destruction. Several candidate antigens have been described recently, including glutamic acid decarboxylase, heat shock protein 65 and peripherin. Restricted T-cell receptor V beta gene usage in non-obese diabetic mice has been suggested but not yet proven. In addition to major histocompatibility genes, several non-H-2 predisposing genes have been mapped on chromosomes 1, 3 and 11.
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Dec
|
| pubmed:issn |
0952-7915
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
3
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
902-5
|
| pubmed:dateRevised |
2005-11-16
|
| pubmed:meshHeading |
pubmed-meshheading:1793533-Autoantigens,
pubmed-meshheading:1793533-Diabetes Mellitus, Type 1,
pubmed-meshheading:1793533-Humans,
pubmed-meshheading:1793533-Immune Tolerance,
pubmed-meshheading:1793533-Immunotherapy,
pubmed-meshheading:1793533-Major Histocompatibility Complex,
pubmed-meshheading:1793533-T-Lymphocytes
|
| pubmed:year |
1991
|
| pubmed:articleTitle |
Insulin-dependent diabetes mellitus.
|
| pubmed:affiliation |
Hôpital Neckar, Paris, France.
|
| pubmed:publicationType |
Journal Article,
Review
|