pubmed-article:17916769 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0004153 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0021665 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C1155266 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
pubmed-article:17916769 | lifeskim:mentions | umls-concept:C0547047 | lld:lifeskim |
pubmed-article:17916769 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:17916769 | pubmed:dateCreated | 2007-11-21 | lld:pubmed |
pubmed-article:17916769 | pubmed:abstractText | Whereas growth factors, via their ability to stimulate vascular smooth muscle cell (VSMC) proliferation and migration, have been thought to play a permissive role in atherosclerosis initiation and progression, the role of insulin-like growth factor-1 (IGF-1) is unknown. Here we report for the first time that IGF-1 infusion decreased atherosclerotic plaque progression in ApoE-deficient mice on a Western diet. | lld:pubmed |
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pubmed-article:17916769 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:language | eng | lld:pubmed |
pubmed-article:17916769 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17916769 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17916769 | pubmed:month | Dec | lld:pubmed |
pubmed-article:17916769 | pubmed:issn | 1524-4636 | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:ShaiShaw-Yung... | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:HigashiYusuke... | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:LiYangxinY | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:DelafontaineP... | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:SongYao-HuaYH | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:SukhanovSergi... | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:MohlerJessica... | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:VaughnCharlot... | lld:pubmed |
pubmed-article:17916769 | pubmed:author | pubmed-author:TitteringtonJ... | lld:pubmed |
pubmed-article:17916769 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:17916769 | pubmed:volume | 27 | lld:pubmed |
pubmed-article:17916769 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17916769 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17916769 | pubmed:pagination | 2684-90 | lld:pubmed |
pubmed-article:17916769 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:17916769 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17916769 | pubmed:articleTitle | IGF-1 reduces inflammatory responses, suppresses oxidative stress, and decreases atherosclerosis progression in ApoE-deficient mice. | lld:pubmed |
pubmed-article:17916769 | pubmed:affiliation | Cardiology Section, Department of Medicine, Tulane University School of Medicine, 1430 Tulane Ave, SL-48, New Orleans, LA 70112, USA. | lld:pubmed |
pubmed-article:17916769 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17916769 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:17916769 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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