Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2007-11-16
pubmed:abstractText
We examined the influence of chronic treatment with angiotensin-(1-7) [Ang-(1-7)] on renox (renal NADPH oxidase, NOX-4) and the development of renal dysfunction in streptozotocin-treated spontaneously hypertensive rats (diabetic SHR).
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1421-9670
pubmed:author
pubmed:copyrightInfo
(c) 2007 S. Karger AG, Basel.
pubmed:issnType
Electronic
pubmed:volume
28
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
25-33
pubmed:meshHeading
pubmed-meshheading:17890855-Angiotensin I, pubmed-meshheading:17890855-Animals, pubmed-meshheading:17890855-Antihypertensive Agents, pubmed-meshheading:17890855-Blood Pressure, pubmed-meshheading:17890855-Body Weight, pubmed-meshheading:17890855-Diabetes Mellitus, Experimental, pubmed-meshheading:17890855-Diabetic Nephropathies, pubmed-meshheading:17890855-Enzyme Activation, pubmed-meshheading:17890855-Gene Expression Regulation, Enzymologic, pubmed-meshheading:17890855-Hyperglycemia, pubmed-meshheading:17890855-Hypertension, Renal, pubmed-meshheading:17890855-Male, pubmed-meshheading:17890855-NADPH Oxidase, pubmed-meshheading:17890855-Peptide Fragments, pubmed-meshheading:17890855-Proteinuria, pubmed-meshheading:17890855-Rats, pubmed-meshheading:17890855-Rats, Inbred SHR, pubmed-meshheading:17890855-Rats, Inbred WKY, pubmed-meshheading:17890855-Renal Circulation, pubmed-meshheading:17890855-Vasoconstriction, pubmed-meshheading:17890855-Vasodilation
pubmed:year
2008
pubmed:articleTitle
Angiotensin-(1-7) prevents activation of NADPH oxidase and renal vascular dysfunction in diabetic hypertensive rats.
pubmed:affiliation
Department of Pharmacology and Toxicology, Faculty of Medicine, Kuwait University, Kuwait. ibenter@hsc.edu.kw
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural