rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
9
|
pubmed:dateCreated |
2007-9-26
|
pubmed:abstractText |
Degeneration of extracellular matrix of cartilage leads to the production of molecules capable of activating the immune system via Toll-like receptor 4 (TLR-4). The objective of this study was to investigate the involvement of TLR-4 activation in the development and progression of autoimmune destructive arthritis.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Sep
|
pubmed:issn |
0004-3591
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
56
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
2957-67
|
pubmed:meshHeading |
pubmed-meshheading:17763416-Animals,
pubmed-meshheading:17763416-Arthritis,
pubmed-meshheading:17763416-Autoimmune Diseases,
pubmed-meshheading:17763416-Escherichia coli,
pubmed-meshheading:17763416-Lipopolysaccharides,
pubmed-meshheading:17763416-Male,
pubmed-meshheading:17763416-Mice,
pubmed-meshheading:17763416-Mice, Inbred BALB C,
pubmed-meshheading:17763416-Mice, Inbred DBA,
pubmed-meshheading:17763416-Toll-Like Receptor 4
|
pubmed:year |
2007
|
pubmed:articleTitle |
Inhibition of Toll-like receptor 4 breaks the inflammatory loop in autoimmune destructive arthritis.
|
pubmed:affiliation |
Radboud University Nijmegen Medical Centre, Nijmegen, The Netherlands. s.abdollahi-roodsaz@reuma.umcn.nl
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|