Source:http://linkedlifedata.com/resource/pubmed/id/17761782
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rdf:type | |
lifeskim:mentions |
umls-concept:C0001480,
umls-concept:C0015127,
umls-concept:C0023078,
umls-concept:C0026336,
umls-concept:C0034693,
umls-concept:C0034721,
umls-concept:C0034929,
umls-concept:C0162772,
umls-concept:C0232483,
umls-concept:C0332282,
umls-concept:C0442805,
umls-concept:C1314792,
umls-concept:C1879547,
umls-concept:C2349975,
umls-concept:C2936400
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pubmed:dateCreated |
2011-1-4
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pubmed:abstractText |
The pathogenetic mechanisms of enhanced laryngeal reflex reactivity (ELRR) in patients with extraoesophageal reflux (EOR) are unclear. In a rat EOR model, a laryngeal acid-pepsin insult produces an ELRR that is mediated through sensitization of the capsaicin-sensitive laryngeal afferent fibers by reactive oxygen species (ROS).
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pubmed:commentsCorrections | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:status |
PubMed-not-MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
1468-3288
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:day |
2
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:year |
2007
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pubmed:articleTitle |
Activation of P2X purinoceptors by ATP subsequent to an increase in reactive oxygen species causes enhanced laryngeal reflex reactivity in a rat model of extraoesophageal reflux.
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pubmed:affiliation |
School of Medicine, National Yang-Ming University, Taiwan.
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pubmed:publicationType |
Journal Article,
Retracted Publication
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