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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
11
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pubmed:dateCreated |
2007-9-11
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pubmed:abstractText |
The endoplasmic reticulum (ER) responds to various stresses by upregulation of ER chaperones, but prolonged ER stress eventually causes apoptosis. Although apoptosis is considered to be essential for the progression and rupture of atherosclerotic plaques, the influence of ER stress and apoptosis on rupture of unstable coronary plaques remains unclear.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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|
pubmed:citationSubset |
AIM
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|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
Sep
|
|
pubmed:issn |
1524-4539
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pubmed:author |
pubmed-author:AsadaYujiroY,
pubmed-author:Bochaton-PiallatMarie-LuceML,
pubmed-author:GabbianiGiulioG,
pubmed-author:HaoHiroyukiH,
pubmed-author:HatakeyamaKintaK,
pubmed-author:Ishibashi-UedaHatsueH,
pubmed-author:KitakazeMasafumiM,
pubmed-author:MinaminoTetsuoT,
pubmed-author:MochizukiNaokiN,
pubmed-author:MyoishiMasafumiM,
pubmed-author:NishihiraKensakuK,
pubmed-author:OkadaKen-ichiroK,
pubmed-author:WatanabeKoukiK
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pubmed:issnType |
Electronic
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pubmed:day |
11
|
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pubmed:volume |
116
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
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pubmed:pagination |
1226-33
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
|
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pubmed:year |
2007
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pubmed:articleTitle |
Increased endoplasmic reticulum stress in atherosclerotic plaques associated with acute coronary syndrome.
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pubmed:affiliation |
Department of Cardiovascular Medicine, National Cardiovascular Center, Suita, Osaka 565-8565, Japan.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
|
