| pubmed-article:17671648 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C1332717 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C1706438 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C0085358 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C0042210 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C1413244 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C2698600 | lld:lifeskim |
| pubmed-article:17671648 | lifeskim:mentions | umls-concept:C1514873 | lld:lifeskim |
| pubmed-article:17671648 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:17671648 | pubmed:dateCreated | 2007-8-2 | lld:pubmed |
| pubmed-article:17671648 | pubmed:abstractText | MHC class I-restricted CD8(+) T cells are necessary to mount an immune response against Mycobacterium tuberculosis. M. tuberculosis antigens can enter MHC class I cross-processing pathways through a number of different mechanisms, including via the uptake of antigen-containing apoptotic vesicles released by infected cells. A study in this issue of the JCI by Hinchey and colleagues shows that M. tuberculosis inhibits host cell apoptosis and thus may interfere with optimal cross-priming and action of CD8(+) T cells (see the related article beginning on page 2279). M. tuberculosis genetically modified to induce apoptosis is shown to be more effective in priming CD8(+) T cells in vivo and therefore may be a more effective vaccine against tuberculosis than the currently utilized M. bovis BCG vaccine. | lld:pubmed |
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| pubmed-article:17671648 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17671648 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17671648 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:17671648 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17671648 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:17671648 | pubmed:issn | 0021-9738 | lld:pubmed |
| pubmed-article:17671648 | pubmed:author | pubmed-author:BoomW HenryWH | lld:pubmed |
| pubmed-article:17671648 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17671648 | pubmed:volume | 117 | lld:pubmed |
| pubmed-article:17671648 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17671648 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17671648 | pubmed:pagination | 2092-4 | lld:pubmed |
| pubmed-article:17671648 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:17671648 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17671648 | pubmed:articleTitle | New TB vaccines: is there a requirement for CD8 T cells? | lld:pubmed |
| pubmed-article:17671648 | pubmed:affiliation | Tuberculosis Research Unit, Case Western Reserve University and University Hospitals' Case Medical Center, Cleveland, Ohio 44106-4984, USA. whb@case.edu | lld:pubmed |
| pubmed-article:17671648 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17671648 | pubmed:publicationType | Comment | lld:pubmed |
| pubmed-article:17671648 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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