Linked Life Data

17671396

Source:http://linkedlifedata.com/resource/pubmed/id/17671396

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2007-9-6
pubmed:abstractText
Fibrosis is a hallmark of progressive organ disease. The 10-kDa interferon-inducible protein IP-10/CXCL10 is a potent chemoattractant for activated T lymphocytes, natural killer cells, and monocytes. However, the involvement of IP-10 in the pathogenesis of renal diseases via its receptor, CXCR3, remains unclear. To contribute to the clarification of this issue was the aim of this study.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1660-2129
pubmed:author
pubmed:copyrightInfo
Copyright 2007 S. Karger AG, Basel.
pubmed:issnType
Electronic
pubmed:volume
107
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
e12-21
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed-meshheading:17671396-Animals, pubmed-meshheading:17671396-Antibodies, Monoclonal, pubmed-meshheading:17671396-Cells, Cultured, pubmed-meshheading:17671396-Chemokine CXCL10, pubmed-meshheading:17671396-Chemokines, CXC, pubmed-meshheading:17671396-Disease Progression, pubmed-meshheading:17671396-Down-Regulation, pubmed-meshheading:17671396-Fibrosis, pubmed-meshheading:17671396-Hepatocyte Growth Factor, pubmed-meshheading:17671396-Hydroxyproline, pubmed-meshheading:17671396-Kidney, pubmed-meshheading:17671396-Kidney Diseases, pubmed-meshheading:17671396-Male, pubmed-meshheading:17671396-Mice, pubmed-meshheading:17671396-Mice, Inbred BALB C, pubmed-meshheading:17671396-Mice, Knockout, pubmed-meshheading:17671396-Receptors, CXCR3, pubmed-meshheading:17671396-Receptors, Chemokine, pubmed-meshheading:17671396-Transforming Growth Factor beta1, pubmed-meshheading:17671396-Up-Regulation, pubmed-meshheading:17671396-Ureteral Obstruction
pubmed:year
2007
pubmed:articleTitle
Blockade of IP-10/CXCR3 promotes progressive renal fibrosis.
pubmed:affiliation
Department of Disease Control and Homeostasis, Kanazawa University Graduate School of Medical Science, Kanazawa, Japan.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't