Source:http://linkedlifedata.com/resource/pubmed/id/17670642
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
5
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pubmed:dateCreated |
2007-8-2
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pubmed:abstractText |
A major limitation of currently available bioprosthetic valves is their propensity to calcify. At present, one approach in tissue-engineering, uses decellularized, xenogenic scaffolds that are implanted, with the expectation of complete matrix repopulation in vivo. Whether or not such a decellularized matrix will be sufficiently endowed to prevent calcification is unknown.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:status |
PubMed-not-MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
1569-9285
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
5
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
544-9
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pubmed:year |
2006
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pubmed:articleTitle |
Acellular porcine and kangaroo aortic valve scaffolds show more intense immune-mediated calcification than cross-linked Toronto SPV valves in the sheep model.
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pubmed:affiliation |
Department of Cardiac Surgery, 5K12, University Hospital Ghent, De Pintelaan 185, BE-9000 Ghent, Belgium. guido.vannooten@ugent.be
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pubmed:publicationType |
Journal Article
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