Linked Life Data

17667714

Source:http://linkedlifedata.com/resource/pubmed/id/17667714

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2007-8-1
pubmed:abstractText
The dysregulation of adaptive immunity is extensively investigated in celiac disease (CD). Recent data also suggest, however, the implication of innate immunity in CD. Toll-like receptors (TLRs) play a central role in the initiation or maintenance of innate immune responses. The aim of this study was to characterise the expression of TLR2, TLR3, and TLR4 in duodenal biopsy samples taken from children with CD and from controls.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Aug
pubmed:issn
1536-4801
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
45
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
187-93
pubmed:dateRevised
2008-11-21
pubmed:meshHeading
pubmed-meshheading:17667714-Adolescent, pubmed-meshheading:17667714-Blotting, Western, pubmed-meshheading:17667714-Case-Control Studies, pubmed-meshheading:17667714-Celiac Disease, pubmed-meshheading:17667714-Child, pubmed-meshheading:17667714-Child, Preschool, pubmed-meshheading:17667714-Female, pubmed-meshheading:17667714-Gene Expression, pubmed-meshheading:17667714-Humans, pubmed-meshheading:17667714-Immunity, Innate, pubmed-meshheading:17667714-Immunohistochemistry, pubmed-meshheading:17667714-Intestinal Mucosa, pubmed-meshheading:17667714-Male, pubmed-meshheading:17667714-RNA, Messenger, pubmed-meshheading:17667714-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:17667714-Toll-Like Receptor 2, pubmed-meshheading:17667714-Toll-Like Receptor 3, pubmed-meshheading:17667714-Toll-Like Receptor 4, pubmed-meshheading:17667714-Toll-Like Receptors, pubmed-meshheading:17667714-Up-Regulation
pubmed:year
2007
pubmed:articleTitle
Increased mucosal expression of Toll-like receptor (TLR)2 and TLR4 in coeliac disease.
pubmed:affiliation
1st Department of Pediatrics, Semmelweis University, Budapest, Hungary. szebeni@gyer1.sote.hu
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't