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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
8
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pubmed:dateCreated |
2007-7-26
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pubmed:abstractText |
Aortic regurgitation (AR) causes left ventricular (LV) volume overload, leading to progressive LV dilatation and dysfunction. In the present study it was examined whether blockade of angiotensin II type 1 receptor (AT1) could improve survival in cases of chronic severe AR.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
1346-9843
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pubmed:author |
pubmed-author:AdachiYuichiroY,
pubmed-author:FukamizuAkiyoshiA,
pubmed-author:HaradaMasakiM,
pubmed-author:KawakamiRikaR,
pubmed-author:KinoshitaHideyukiH,
pubmed-author:KishimotoIchiroI,
pubmed-author:KuwaharaKoichiroK,
pubmed-author:NakagawaYasuakiY,
pubmed-author:NakanishiMichioM,
pubmed-author:NakaoKazuwaK,
pubmed-author:SaitoYoshihikoY,
pubmed-author:UsamiSatoruS,
pubmed-author:YasunoShinjiS
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pubmed:issnType |
Print
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pubmed:volume |
71
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1310-6
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:17652901-Animals,
pubmed-meshheading:17652901-Aortic Valve Insufficiency,
pubmed-meshheading:17652901-Chronic Disease,
pubmed-meshheading:17652901-Disease Models, Animal,
pubmed-meshheading:17652901-Heart Failure,
pubmed-meshheading:17652901-Mice,
pubmed-meshheading:17652901-Mice, Knockout,
pubmed-meshheading:17652901-Natriuretic Peptide, Brain,
pubmed-meshheading:17652901-RNA, Messenger,
pubmed-meshheading:17652901-Receptor, Angiotensin, Type 1,
pubmed-meshheading:17652901-Survival Rate,
pubmed-meshheading:17652901-Ventricular Dysfunction, Left,
pubmed-meshheading:17652901-Ventricular Remodeling
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pubmed:year |
2007
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pubmed:articleTitle |
Genetic disruption of angiotensin II type 1a receptor improves long-term survival of mice with chronic severe aortic regurgitation.
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pubmed:affiliation |
Department of Medicine and Clinical Science, Kyoto University Graduate School of Medicine, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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