| pubmed-article:17641165 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C1167395 | lld:lifeskim |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
| pubmed-article:17641165 | lifeskim:mentions | umls-concept:C0205164 | lld:lifeskim |
| pubmed-article:17641165 | pubmed:issue | 5840 | lld:pubmed |
| pubmed-article:17641165 | pubmed:dateCreated | 2007-8-17 | lld:pubmed |
| pubmed-article:17641165 | pubmed:abstractText | Understanding why some people establish and maintain effective control of HIV-1 and others do not is a priority in the effort to develop new treatments for HIV/AIDS. Using a whole-genome association strategy, we identified polymorphisms that explain nearly 15% of the variation among individuals in viral load during the asymptomatic set-point period of infection. One of these is found within an endogenous retroviral element and is associated with major histocompatibility allele human leukocyte antigen (HLA)-B*5701, whereas a second is located near the HLA-C gene. An additional analysis of the time to HIV disease progression implicated two genes, one of which encodes an RNA polymerase I subunit. These findings emphasize the importance of studying human genetic variation as a guide to combating infectious agents. | lld:pubmed |
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| pubmed-article:17641165 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17641165 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17641165 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17641165 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17641165 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:17641165 | pubmed:issn | 1095-9203 | lld:pubmed |
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| pubmed-article:17641165 | pubmed:author | pubmed-author:MiroJosé MJM | lld:pubmed |
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| pubmed-article:17641165 | pubmed:author | pubmed-author:WealeMikeM | lld:pubmed |
| pubmed-article:17641165 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:17641165 | pubmed:day | 17 | lld:pubmed |
| pubmed-article:17641165 | pubmed:volume | 317 | lld:pubmed |
| pubmed-article:17641165 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17641165 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17641165 | pubmed:pagination | 944-7 | lld:pubmed |
| pubmed-article:17641165 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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