| pubmed-article:17620961 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17620961 | lifeskim:mentions | umls-concept:C0178719 | lld:lifeskim |
| pubmed-article:17620961 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:17620961 | lifeskim:mentions | umls-concept:C0521942 | lld:lifeskim |
| pubmed-article:17620961 | lifeskim:mentions | umls-concept:C0439807 | lld:lifeskim |
| pubmed-article:17620961 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:17620961 | pubmed:dateCreated | 2007-7-10 | lld:pubmed |
| pubmed-article:17620961 | pubmed:abstractText | Angiotensin II plays a crucial role in the induction of oxidative stress and the pathogenesis of cardiovascular and renal diseases, and the beneficial mechanisms of angiotensin II receptor 1 blockers (ARBs) are multifactorial. We investigated the receptor-independent protective role of an ARB using primary-cultured mesangial cells from angiotensin II receptor 1 knockout or wild-type mice and a highly lipophilic ARB, telmisartan. | lld:pubmed |
| pubmed-article:17620961 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17620961 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17620961 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17620961 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:17620961 | pubmed:issn | 0263-6352 | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:FujitaToshiro... | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:NangakuMasaom... | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:InagiReikoR | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:MiyataToshioT | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:MatsusakaTaij... | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:KatoHidekiH | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:NoiriEiseiE | lld:pubmed |
| pubmed-article:17620961 | pubmed:author | pubmed-author:ShaoJingJ | lld:pubmed |
| pubmed-article:17620961 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17620961 | pubmed:volume | 25 | lld:pubmed |
| pubmed-article:17620961 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17620961 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17620961 | pubmed:pagination | 1643-9 | lld:pubmed |
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| pubmed-article:17620961 | pubmed:meshHeading | pubmed-meshheading:17620961... | lld:pubmed |
| pubmed-article:17620961 | pubmed:meshHeading | pubmed-meshheading:17620961... | lld:pubmed |
| pubmed-article:17620961 | pubmed:meshHeading | pubmed-meshheading:17620961... | lld:pubmed |
| pubmed-article:17620961 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17620961 | pubmed:articleTitle | Receptor-independent intracellular radical scavenging activity of an angiotensin II receptor blocker. | lld:pubmed |
| pubmed-article:17620961 | pubmed:affiliation | Division of Nephrology and Endocrinology, University of Tokyo School of Medicine, Tokyo, Japan. | lld:pubmed |
| pubmed-article:17620961 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17620961 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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