|
rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
3
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|
pubmed:dateCreated |
2007-7-6
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|
pubmed:abstractText |
To investigate effect of pinacidil, an ATP sensitive potassium channel (K(ATP)) opener, on the neuronal apoptosis and its signaling transduction mechanism following focal cerebral ischemia-reperfusion in rats.
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pubmed:language |
eng
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pubmed:journal |
|
|
pubmed:citationSubset |
IM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
May
|
|
pubmed:issn |
1673-7067
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|
pubmed:author |
|
|
pubmed:issnType |
Print
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|
pubmed:volume |
23
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
145-50
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pubmed:meshHeading |
pubmed-meshheading:17612592-Animals,
pubmed-meshheading:17612592-Antihypertensive Agents,
pubmed-meshheading:17612592-Apoptosis,
pubmed-meshheading:17612592-Brain Ischemia,
pubmed-meshheading:17612592-Caspases,
pubmed-meshheading:17612592-Gene Expression Regulation,
pubmed-meshheading:17612592-In Situ Nick-End Labeling,
pubmed-meshheading:17612592-Male,
pubmed-meshheading:17612592-Neurons,
pubmed-meshheading:17612592-Pinacidil,
pubmed-meshheading:17612592-RNA, Messenger,
pubmed-meshheading:17612592-Rats,
pubmed-meshheading:17612592-Rats, Wistar,
pubmed-meshheading:17612592-Reperfusion Injury,
pubmed-meshheading:17612592-Time Factors
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|
pubmed:year |
2007
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|
pubmed:articleTitle |
Pinacidil reduces neuronal apoptosis following cerebral ischemia-reperfusion in rats through both mitochondrial and death-receptor signal pathways.
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|
pubmed:affiliation |
Department of Neurology, the Affiliated Shengjing Hospital, China Medical University, Shenyang 110004, China.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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