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pubmed-article:17586410pubmed:abstractTextReduced synthesis of nitric oxide (NO) is considered a major proatherogenic mechanism in patients with end-stage renal disease (ESRD), but genetic factors impinging on this mechanism have been little studied in this population.lld:pubmed
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pubmed-article:17586410pubmed:articleTitleAn additive effect of endothelial nitric oxide synthase gene polymorphisms contributes to the severity of atherosclerosis in patients on dialysis.lld:pubmed
pubmed-article:17586410pubmed:affiliationCNR-IBIM, Institute of Biomedicine, Clinical Epidemiology and Physiopathology of Renal Diseases and Hypertension & Division of Nephrology, Reggio Calabria, Italy.lld:pubmed
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