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rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
7
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|
pubmed:dateCreated |
2007-6-25
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|
pubmed:abstractText |
Reduced synthesis of nitric oxide (NO) is considered a major proatherogenic mechanism in patients with end-stage renal disease (ESRD), but genetic factors impinging on this mechanism have been little studied in this population.
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|
pubmed:language |
eng
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|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
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|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Jul
|
|
pubmed:issn |
0895-7061
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|
pubmed:author |
|
|
pubmed:issnType |
Print
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|
pubmed:volume |
20
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
758-63
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|
pubmed:dateRevised |
2009-2-24
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|
pubmed:meshHeading |
pubmed-meshheading:17586410-Adult,
pubmed-meshheading:17586410-Aged,
pubmed-meshheading:17586410-Alleles,
pubmed-meshheading:17586410-Atherosclerosis,
pubmed-meshheading:17586410-Carotid Artery, Common,
pubmed-meshheading:17586410-Female,
pubmed-meshheading:17586410-Humans,
pubmed-meshheading:17586410-Italy,
pubmed-meshheading:17586410-Kidney Failure, Chronic,
pubmed-meshheading:17586410-Male,
pubmed-meshheading:17586410-Middle Aged,
pubmed-meshheading:17586410-Nitric Oxide Synthase Type III,
pubmed-meshheading:17586410-Polymorphism, Genetic,
pubmed-meshheading:17586410-Regression Analysis,
pubmed-meshheading:17586410-Renal Dialysis,
pubmed-meshheading:17586410-Risk Factors,
pubmed-meshheading:17586410-Tunica Intima,
pubmed-meshheading:17586410-Tunica Media
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|
pubmed:year |
2007
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|
pubmed:articleTitle |
An additive effect of endothelial nitric oxide synthase gene polymorphisms contributes to the severity of atherosclerosis in patients on dialysis.
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|
pubmed:affiliation |
CNR-IBIM, Institute of Biomedicine, Clinical Epidemiology and Physiopathology of Renal Diseases and Hypertension & Division of Nephrology, Reggio Calabria, Italy.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|
