rdf:type |
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lifeskim:mentions |
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pubmed:issue |
24
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pubmed:dateCreated |
2007-6-19
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pubmed:abstractText |
In a recent study, we demonstrated that an increase in oxidative stress in the rostral ventrolateral medulla plays a critical role in the sympathoexcitation observed in chronic heart failure (CHF). Growing evidence indicates that exercise training evokes an antioxidative effect in CHF. In the present study, we therefore hypothesized that long-term exercise exerts its beneficial effect on autonomic activity in CHF via central antioxidative mechanisms.
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pubmed:grant |
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
AIM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
1524-4539
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pubmed:author |
|
pubmed:issnType |
Electronic
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pubmed:day |
19
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pubmed:volume |
115
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
3095-102
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:17548725-Animals,
pubmed-meshheading:17548725-Antioxidants,
pubmed-meshheading:17548725-Baroreflex,
pubmed-meshheading:17548725-Blotting, Western,
pubmed-meshheading:17548725-Body Weight,
pubmed-meshheading:17548725-Chronic Disease,
pubmed-meshheading:17548725-Disease Models, Animal,
pubmed-meshheading:17548725-Echocardiography,
pubmed-meshheading:17548725-Exercise Therapy,
pubmed-meshheading:17548725-Free Radicals,
pubmed-meshheading:17548725-Gene Expression Regulation, Enzymologic,
pubmed-meshheading:17548725-Gene Transfer Techniques,
pubmed-meshheading:17548725-Heart Failure,
pubmed-meshheading:17548725-Male,
pubmed-meshheading:17548725-Myocardium,
pubmed-meshheading:17548725-NADPH Oxidase,
pubmed-meshheading:17548725-Organ Size,
pubmed-meshheading:17548725-Oxidative Stress,
pubmed-meshheading:17548725-Pacemaker, Artificial,
pubmed-meshheading:17548725-Rabbits,
pubmed-meshheading:17548725-Superoxide Dismutase,
pubmed-meshheading:17548725-Sympathetic Nervous System
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pubmed:year |
2007
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pubmed:articleTitle |
Exercise training normalizes sympathetic outflow by central antioxidant mechanisms in rabbits with pacing-induced chronic heart failure.
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pubmed:affiliation |
Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, 985850 Nebraska Medical Center, Omaha, NE 68198-5850, USA. lgao@unmc.edu
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
|