Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
24
pubmed:dateCreated
2007-6-19
pubmed:abstractText
In a recent study, we demonstrated that an increase in oxidative stress in the rostral ventrolateral medulla plays a critical role in the sympathoexcitation observed in chronic heart failure (CHF). Growing evidence indicates that exercise training evokes an antioxidative effect in CHF. In the present study, we therefore hypothesized that long-term exercise exerts its beneficial effect on autonomic activity in CHF via central antioxidative mechanisms.
pubmed:grant
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jun
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
19
pubmed:volume
115
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
3095-102
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed-meshheading:17548725-Animals, pubmed-meshheading:17548725-Antioxidants, pubmed-meshheading:17548725-Baroreflex, pubmed-meshheading:17548725-Blotting, Western, pubmed-meshheading:17548725-Body Weight, pubmed-meshheading:17548725-Chronic Disease, pubmed-meshheading:17548725-Disease Models, Animal, pubmed-meshheading:17548725-Echocardiography, pubmed-meshheading:17548725-Exercise Therapy, pubmed-meshheading:17548725-Free Radicals, pubmed-meshheading:17548725-Gene Expression Regulation, Enzymologic, pubmed-meshheading:17548725-Gene Transfer Techniques, pubmed-meshheading:17548725-Heart Failure, pubmed-meshheading:17548725-Male, pubmed-meshheading:17548725-Myocardium, pubmed-meshheading:17548725-NADPH Oxidase, pubmed-meshheading:17548725-Organ Size, pubmed-meshheading:17548725-Oxidative Stress, pubmed-meshheading:17548725-Pacemaker, Artificial, pubmed-meshheading:17548725-Rabbits, pubmed-meshheading:17548725-Superoxide Dismutase, pubmed-meshheading:17548725-Sympathetic Nervous System
pubmed:year
2007
pubmed:articleTitle
Exercise training normalizes sympathetic outflow by central antioxidant mechanisms in rabbits with pacing-induced chronic heart failure.
pubmed:affiliation
Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, 985850 Nebraska Medical Center, Omaha, NE 68198-5850, USA. lgao@unmc.edu
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural