Linked Life Data

17511035

Source:http://linkedlifedata.com/resource/pubmed/id/17511035

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
16
pubmed:dateCreated
2007-5-18
pubmed:abstractText
To delineate the mechanisms of renal vasocon-striction in hepatorenal syndrome (HRS), we investigated the expression of type I inositol 1, 4, 5-triphosphate receptors (IP3RI) of kidney in mice with fulminant hepatic failure (FHF).
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
1007-9327
pubmed:author
pubmed:issnType
Print
pubmed:day
28
pubmed:volume
13
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2344-8
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:17511035-Animals, pubmed-meshheading:17511035-Calcium Channels, pubmed-meshheading:17511035-Disease Models, Animal, pubmed-meshheading:17511035-Disease Progression, pubmed-meshheading:17511035-Galactosamine, pubmed-meshheading:17511035-Glomerular Mesangium, pubmed-meshheading:17511035-Hepatorenal Syndrome, pubmed-meshheading:17511035-Inositol 1,4,5-Trisphosphate Receptors, pubmed-meshheading:17511035-Kidney, pubmed-meshheading:17511035-Lipopolysaccharides, pubmed-meshheading:17511035-Liver Failure, Acute, pubmed-meshheading:17511035-Male, pubmed-meshheading:17511035-Membrane Glycoproteins, pubmed-meshheading:17511035-Mice, pubmed-meshheading:17511035-Mice, Inbred BALB C, pubmed-meshheading:17511035-Muscle, Smooth, Vascular, pubmed-meshheading:17511035-RNA, Messenger, pubmed-meshheading:17511035-Receptors, Cytoplasmic and Nuclear, pubmed-meshheading:17511035-Up-Regulation
pubmed:year
2007
pubmed:articleTitle
Type I inositol 1, 4, 5-triphosphate receptors increase in kidney of mice with fulminant hepatic failure.
pubmed:affiliation
Department of Infectious Diseases, The First Affiliated Hospital, China Medical University, No. 155 Nanjing North Street, Shenyang 110001, Liaoning Province, China.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't