17502225

Source:http://linkedlifedata.com/resource/pubmed/id/17502225

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0006826, umls-concept:C0035820, umls-concept:C0242358, umls-concept:C0596290
pubmed:issue	1
pubmed:dateCreated	2007-5-15
pubmed:abstractText	The acquisition of a cancerous phenotype by epithelial cells involves the disruption of intercellular adhesions. The reorganization of the E-cadherin/beta-catenin complex in adherens junctions during cell transformation is widely recognized. Instead the implication of tight junctions (TJs) in this process is starting to be unraveled. The aim of this article is to review the role of TJ proteins in cell proliferation and cancer.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0253725
pubmed:citationSubset	IM
pubmed:status	MEDLINE
pubmed:issn	0079-6336
pubmed:author	pubmed-author:GarayErikaE, pubmed-author:González-MariscalLorenzaL, pubmed-author:LechugaSusanaS
pubmed:issnType	Print
pubmed:volume	42
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	1-57
pubmed:meshHeading	pubmed-meshheading:17502225-Cell Proliferation, pubmed-meshheading:17502225-Models, Biological, pubmed-meshheading:17502225-Neoplasms, pubmed-meshheading:17502225-Tight Junctions
pubmed:year	2007
pubmed:articleTitle	Role of tight junctions in cell proliferation and cancer.
pubmed:affiliation	Center for Research and Advanced Studies (Cinvestav), Department of Physiology, Biophysics and Neuroscience, Ave. Instituto Politécnico Nacional 2508, México, DF 07360, México. lorenza@fisio.cinvestav.mx
pubmed:publicationType	Journal Article, Review, Research Support, Non-U.S. Gov't