| pubmed-article:17475315 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C0002658 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C0001554 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C0080194 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C0871125 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C1705241 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C0205373 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C0332453 | lld:lifeskim |
| pubmed-article:17475315 | lifeskim:mentions | umls-concept:C1705242 | lld:lifeskim |
| pubmed-article:17475315 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:17475315 | pubmed:dateCreated | 2007-6-8 | lld:pubmed |
| pubmed-article:17475315 | pubmed:abstractText | Sprague Dawley (SD) rats are significantly more sensitive than Long Evans (LE) rats to the disruption of prepulse inhibition (PPI) by systemically-administered dopamine (DA) agonists. This strain difference is heritable and insensitive to cross-fostering. Inherited differences in the ability of elevated DA activity to disrupt PPI may be useful for understanding the neural basis for PPI deficits in schizophrenia and other neuropsychiatric disorders. | lld:pubmed |
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| pubmed-article:17475315 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:17475315 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17475315 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17475315 | pubmed:month | May | lld:pubmed |
| pubmed-article:17475315 | pubmed:issn | 0091-3057 | lld:pubmed |
| pubmed-article:17475315 | pubmed:author | pubmed-author:SwerdlowNeal... | lld:pubmed |
| pubmed-article:17475315 | pubmed:author | pubmed-author:ShoemakerJody... | lld:pubmed |
| pubmed-article:17475315 | pubmed:author | pubmed-author:Saint... | lld:pubmed |
| pubmed-article:17475315 | pubmed:author | pubmed-author:BongiovanniMi... | lld:pubmed |
| pubmed-article:17475315 | pubmed:author | pubmed-author:NearyAlaina... | lld:pubmed |
| pubmed-article:17475315 | pubmed:author | pubmed-author:TochenLaura... | lld:pubmed |
| pubmed-article:17475315 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17475315 | pubmed:volume | 87 | lld:pubmed |
| pubmed-article:17475315 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17475315 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17475315 | pubmed:pagination | 1-10 | lld:pubmed |
| pubmed-article:17475315 | pubmed:dateRevised | 2011-9-15 | lld:pubmed |
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| pubmed-article:17475315 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17475315 | pubmed:articleTitle | Strain differences in the disruption of prepulse inhibition of startle after systemic and intra-accumbens amphetamine administration. | lld:pubmed |
| pubmed-article:17475315 | pubmed:affiliation | Department of Psychiatry, UCSD School of Medicine, La Jolla, CA 92093-0804, USA. nswerdlow@ucsd.edu | lld:pubmed |
| pubmed-article:17475315 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17475315 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:17475315 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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