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pubmed-article:17473215pubmed:abstractTextPancreatic cancer is a highly aggressive disease that remains refractory to various chemotherapeutic agents. Because the proto-oncogene c-myc can modulate apoptosis in response to cytotoxic insults and is commonly overexpressed in pancreatic cancer, we investigated the value of c-myc as a potential modulator of cellular response to various chemotherapeutic agents.lld:pubmed
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pubmed-article:17473215pubmed:articleTitlec-Myc-induced chemosensitization is mediated by suppression of cyclin D1 expression and nuclear factor-kappa B activity in pancreatic cancer cells.lld:pubmed
pubmed-article:17473215pubmed:affiliationDepartment of Pathology, Wayne State University School of Medicine, Detroit, Michigan, USA.lld:pubmed
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