Source:http://linkedlifedata.com/resource/pubmed/id/17449602
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
5
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pubmed:dateCreated |
2007-4-23
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pubmed:abstractText |
Selenium (Se), an essential trace element, and vitamin E, a lipid soluble antioxidant, are important mediators for protection against oxidative stress. Recent work has demonstrated that deficiencies in either Se or vitamin E result in increased viral pathogenicity and altered immune responses. Furthermore, deficiencies in either Se or vitamin E results in specific viral mutations, changing relatively benign viruses into virulent ones. Thus, host nutritional status should be considered a driving force for the emergence of new viral strains or newly pathogenic strains of known viruses.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
0022-3166
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
137
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1338-40
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pubmed:dateRevised |
2007-12-3
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pubmed:meshHeading |
pubmed-meshheading:17449602-Animals,
pubmed-meshheading:17449602-Antioxidants,
pubmed-meshheading:17449602-Humans,
pubmed-meshheading:17449602-Mutation,
pubmed-meshheading:17449602-Nutritional Status,
pubmed-meshheading:17449602-Selenium,
pubmed-meshheading:17449602-Trace Elements,
pubmed-meshheading:17449602-Viruses,
pubmed-meshheading:17449602-Vitamin E Deficiency
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pubmed:year |
2007
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pubmed:articleTitle |
Selenium and vitamin E status: impact on viral pathogenicity.
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pubmed:affiliation |
Department of Nutrition, University of North Carolina, Chapel Hill, NC 27599-7461, USA. melinda_beck@unc.edu
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pubmed:publicationType |
Journal Article,
Review,
Research Support, N.I.H., Extramural
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