rdf:type |
|
lifeskim:mentions |
umls-concept:C0008312,
umls-concept:C0017262,
umls-concept:C0023810,
umls-concept:C0026473,
umls-concept:C0030705,
umls-concept:C0175697,
umls-concept:C0185117,
umls-concept:C0205349,
umls-concept:C1334043,
umls-concept:C1824676,
umls-concept:C2697656,
umls-concept:C2911684
|
pubmed:issue |
3
|
pubmed:dateCreated |
2007-8-6
|
pubmed:abstractText |
Toll-like receptors (TLRs) have emerged as a key component of the innate immune system that triggers antimicrobial responses. Altered monocyte responses to ligands for TLRs have been reported in patients with primary biliary cirrhosis (PBC), yet the precise mechanism remains unknown.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD,
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD14,
http://linkedlifedata.com/resource/pubmed/chemical/CD180 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/CD83 antigen,
http://linkedlifedata.com/resource/pubmed/chemical/Immunoglobulins,
http://linkedlifedata.com/resource/pubmed/chemical/Ligands,
http://linkedlifedata.com/resource/pubmed/chemical/Lipopolysaccharides,
http://linkedlifedata.com/resource/pubmed/chemical/MYD88 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Membrane Glycoproteins,
http://linkedlifedata.com/resource/pubmed/chemical/Myeloid Differentiation Factor 88,
http://linkedlifedata.com/resource/pubmed/chemical/NF-kappa B,
http://linkedlifedata.com/resource/pubmed/chemical/Peptidoglycan,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger,
http://linkedlifedata.com/resource/pubmed/chemical/TLR2 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Toll-Like Receptor 2,
http://linkedlifedata.com/resource/pubmed/chemical/Toll-Like Receptor 4
|
pubmed:status |
MEDLINE
|
pubmed:month |
Sep
|
pubmed:issn |
0168-8278
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
47
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
404-11
|
pubmed:meshHeading |
pubmed-meshheading:17448566-Aged,
pubmed-meshheading:17448566-Antigens, CD,
pubmed-meshheading:17448566-Antigens, CD14,
pubmed-meshheading:17448566-Cell Membrane,
pubmed-meshheading:17448566-Chronic Disease,
pubmed-meshheading:17448566-Flow Cytometry,
pubmed-meshheading:17448566-Hepatitis, Viral, Human,
pubmed-meshheading:17448566-Humans,
pubmed-meshheading:17448566-Immunoglobulins,
pubmed-meshheading:17448566-Ligands,
pubmed-meshheading:17448566-Lipopolysaccharides,
pubmed-meshheading:17448566-Liver Cirrhosis, Biliary,
pubmed-meshheading:17448566-Membrane Glycoproteins,
pubmed-meshheading:17448566-Middle Aged,
pubmed-meshheading:17448566-Monocytes,
pubmed-meshheading:17448566-Myeloid Differentiation Factor 88,
pubmed-meshheading:17448566-NF-kappa B,
pubmed-meshheading:17448566-Peptidoglycan,
pubmed-meshheading:17448566-RNA, Messenger,
pubmed-meshheading:17448566-Toll-Like Receptor 2,
pubmed-meshheading:17448566-Toll-Like Receptor 4
|
pubmed:year |
2007
|
pubmed:articleTitle |
Altered expression of TLR homolog RP105 on monocytes hypersensitive to LPS in patients with primary biliary cirrhosis.
|
pubmed:affiliation |
Division of Gastroenterology and Hepatology, Niigata University Graduate School of Medical and Dental Sciences, 757, Asahimachi-Dori 1, Niigata 951-8510, Japan.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|