pubmed-article:17428547 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17428547 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:17428547 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:17428547 | lifeskim:mentions | umls-concept:C0027814 | lld:lifeskim |
pubmed-article:17428547 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
pubmed-article:17428547 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
pubmed-article:17428547 | pubmed:issue | 1-2 | lld:pubmed |
pubmed-article:17428547 | pubmed:dateCreated | 2007-5-28 | lld:pubmed |
pubmed-article:17428547 | pubmed:abstractText | The role of tumor necrosis factor (TNF)-alpha and its receptors in the pathogenesis of experimental autoimmune neuritis (EAN) induced by P0 peptide 180-199 in TNFR1 (p55) deficient (TNFR1-/-) mice was investigated. Compared to wild type EAN mice, TNFR1-/- EAN mice developed significantly more severe clinical signs, in parallel with enhanced numbers of inflammatory infiltrating cells in peripheral nerves and splenic P0-reactive T cell proliferation, as well as increased obviously MHC class II and CCR3 expression on the macrophages in the cauda equina. Our data indicated that TNF-alpha might have anti-inflammatory effect preventing the development of EAN in this mouse model. | lld:pubmed |
pubmed-article:17428547 | pubmed:language | eng | lld:pubmed |
pubmed-article:17428547 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17428547 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17428547 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17428547 | pubmed:month | May | lld:pubmed |
pubmed-article:17428547 | pubmed:issn | 0165-5728 | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:LEVILa LLL | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:TANT LTL | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:LjunggrenHans... | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:YangXinX | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:MixEilhardE | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:DuanRui-Sheng... | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:QuezadaHernan... | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:LuMing-OuMO | lld:pubmed |
pubmed-article:17428547 | pubmed:author | pubmed-author:ChenZhi-GuoZG | lld:pubmed |
pubmed-article:17428547 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17428547 | pubmed:volume | 186 | lld:pubmed |
pubmed-article:17428547 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17428547 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17428547 | pubmed:pagination | 19-26 | lld:pubmed |
pubmed-article:17428547 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:17428547 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17428547 | pubmed:articleTitle | Aggravation of experimental autoimmune neuritis in TNF-alpha receptor 1 deficient mice. | lld:pubmed |
pubmed-article:17428547 | pubmed:affiliation | Division of Neurodegeneration and Neuroinflammation, Karolinska Institute, Karolinska University Hospital Huddinge, Stockholm, Sweden. | lld:pubmed |
pubmed-article:17428547 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17428547 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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