rdf:type |
|
lifeskim:mentions |
umls-concept:C0001613,
umls-concept:C0007776,
umls-concept:C0019151,
umls-concept:C0022655,
umls-concept:C0026649,
umls-concept:C0030705,
umls-concept:C0205146,
umls-concept:C0205216,
umls-concept:C0233565,
umls-concept:C0439849,
umls-concept:C0445223,
umls-concept:C0599781,
umls-concept:C1552599,
umls-concept:C1704686,
umls-concept:C1704787,
umls-concept:C2347946
|
pubmed:issue |
6
|
pubmed:dateCreated |
2007-5-7
|
pubmed:abstractText |
Bradykinesia is one of the first symptoms of hepatic encephalopathy (HE). Recently it has been suggested that bradykinesia in HE is due to disturbances in movement initiation. Areas involved in self-initiated movement are the motor- and premotor cortex, the supplementary motor cortex, the motor areas of the cingulate gyrus, and part of the frontomesial- and parietal cortex. The present study aimed to test the hypothesis that bradykinesia in HE is due to a functional disturbance of these areas.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Jun
|
pubmed:issn |
0168-8278
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
46
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1034-9
|
pubmed:meshHeading |
pubmed-meshheading:17400326-Adult,
pubmed-meshheading:17400326-Brain,
pubmed-meshheading:17400326-Female,
pubmed-meshheading:17400326-Fluorodeoxyglucose F18,
pubmed-meshheading:17400326-Glucose,
pubmed-meshheading:17400326-Hepatic Encephalopathy,
pubmed-meshheading:17400326-Humans,
pubmed-meshheading:17400326-Hypokinesia,
pubmed-meshheading:17400326-Imaging, Three-Dimensional,
pubmed-meshheading:17400326-Liver,
pubmed-meshheading:17400326-Liver Cirrhosis,
pubmed-meshheading:17400326-Male,
pubmed-meshheading:17400326-Middle Aged,
pubmed-meshheading:17400326-Motor Cortex,
pubmed-meshheading:17400326-Movement
|
pubmed:year |
2007
|
pubmed:articleTitle |
Bradykinesia in cirrhotic patients with early hepatic encephalopathy is related to a decreased glucose uptake of frontomesial cortical areas relevant for movement initiation.
|
pubmed:affiliation |
Department of Neurology, Medizinische Hochschule Hannover, 30623 Hannover, Germany.
|
pubmed:publicationType |
Journal Article
|