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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2007-11-6
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pubmed:abstractText |
We conducted a prospective study to test the hypothesis that the activation of neutrophil elastase-mediated fibrinolysis is insufficient to overcome the fibrinolytic shutdown of disseminated intravascular coagulation (DIC) in patients associated with systemic inflammation.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0049-3848
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
121
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
67-73
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pubmed:meshHeading |
pubmed-meshheading:17397908-Adult,
pubmed-meshheading:17397908-Disseminated Intravascular Coagulation,
pubmed-meshheading:17397908-Female,
pubmed-meshheading:17397908-Fibrinolysis,
pubmed-meshheading:17397908-Humans,
pubmed-meshheading:17397908-Leukocyte Elastase,
pubmed-meshheading:17397908-Male,
pubmed-meshheading:17397908-Middle Aged,
pubmed-meshheading:17397908-Plasminogen Activator Inhibitor 1,
pubmed-meshheading:17397908-Prospective Studies,
pubmed-meshheading:17397908-Sepsis,
pubmed-meshheading:17397908-Systemic Inflammatory Response Syndrome
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pubmed:year |
2007
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pubmed:articleTitle |
The activation of neutrophil elastase-mediated fibrinolysis is not sufficient to overcome the fibrinolytic shutdown of disseminated intravascular coagulation associated with systemic inflammation.
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pubmed:affiliation |
Department of Anesthesiology and Critical Care Medicine, Hokkaido University Graduate School of Medicine, N17 W5, Kita-ku, Sapporo 060 Japan. gando@med.hokudai.ac.jp
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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