|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0011065,
umls-concept:C0038454,
umls-concept:C0040715,
umls-concept:C0041904,
umls-concept:C0205421,
umls-concept:C0242184,
umls-concept:C0521390,
umls-concept:C0599718,
umls-concept:C0599813,
umls-concept:C0599893,
umls-concept:C1412812,
umls-concept:C1522702
|
|
pubmed:issue |
5
|
|
pubmed:dateCreated |
2007-4-24
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|
pubmed:abstractText |
Delayed neuronal death is a hallmark feature of stroke and the primary target of neuroprotective strategies. Caspase-independent apoptosis pathways are suggested as a mechanism for the delayed neuronal injury. Here we test the hypothesis that one of the caspase-independent apoptosis pathways is activated by BNIP3 and mediated by EndoG.
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|
pubmed:language |
eng
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|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
May
|
|
pubmed:issn |
1524-4628
|
|
pubmed:author |
|
|
pubmed:issnType |
Electronic
|
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pubmed:volume |
38
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
1606-13
|
|
pubmed:dateRevised |
2007-11-15
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|
pubmed:meshHeading |
pubmed-meshheading:17379825-Animals,
pubmed-meshheading:17379825-Apoptosis,
pubmed-meshheading:17379825-Disease Models, Animal,
pubmed-meshheading:17379825-Hypoxia, Brain,
pubmed-meshheading:17379825-Male,
pubmed-meshheading:17379825-Membrane Proteins,
pubmed-meshheading:17379825-Mitochondrial Proteins,
pubmed-meshheading:17379825-Neurons,
pubmed-meshheading:17379825-Proto-Oncogene Proteins,
pubmed-meshheading:17379825-Rats,
pubmed-meshheading:17379825-Rats, Sprague-Dawley,
pubmed-meshheading:17379825-Stroke,
pubmed-meshheading:17379825-Translocation, Genetic,
pubmed-meshheading:17379825-Up-Regulation
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|
pubmed:year |
2007
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|
pubmed:articleTitle |
BNIP3 upregulation and EndoG translocation in delayed neuronal death in stroke and in hypoxia.
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|
pubmed:affiliation |
Department of Human Anatomy and Cell Science, University of Manitoba, Faculty of Medicine, Winnipeg, Manitoba, Canada.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|
