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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
2
|
| pubmed:dateCreated |
1992-3-11
|
| pubmed:abstractText |
Chronic supraventricular tachycardia (SVT) causes a dilated cardiomyopathy and myocyte injury. Termination of SVT improves left ventricular (LV) function but is associated with LV hypertrophy. Changes in myocardial blood flow (MBF) that may accompany the development of and recovery from SVT cardiomyopathy might have a significant effect on LV function and myocyte structure. The goal of this study was to relate changes in LV function, myocyte composition, and coronary vascular structure to changes in MBF with the development and recovery of SVT cardiomyopathy.
|
| pubmed:grant | |
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
AIM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Feb
|
| pubmed:issn |
0009-7322
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
85
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
717-29
|
| pubmed:dateRevised |
2010-3-24
|
| pubmed:meshHeading |
pubmed-meshheading:1735164-Adenosine,
pubmed-meshheading:1735164-Animals,
pubmed-meshheading:1735164-Arterioles,
pubmed-meshheading:1735164-Capillaries,
pubmed-meshheading:1735164-Cardiac Pacing, Artificial,
pubmed-meshheading:1735164-Cardiomyopathies,
pubmed-meshheading:1735164-Coronary Circulation,
pubmed-meshheading:1735164-Myocardium,
pubmed-meshheading:1735164-Swine,
pubmed-meshheading:1735164-Tachycardia,
pubmed-meshheading:1735164-Ventricular Function
|
| pubmed:year |
1992
|
| pubmed:articleTitle |
Changes in myocardial blood flow during development of and recovery from tachycardia-induced cardiomyopathy.
|
| pubmed:affiliation |
Division of Cardiothoracic Surgery, Medical University of South Carolina, Charleston 29425.
|
| pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, U.S. Gov't, Non-P.H.S.,
Research Support, Non-U.S. Gov't
|