Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2007-5-2
pubmed:abstractText
The pathogenesis of exercise-induced bronchoconstriction (EIB) involves the release of mediators from several airway cells in response to exercise challenge, but the mechanism leading to airflow obstruction during EIB is incompletely understood.
pubmed:grant
pubmed:commentsCorrections
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pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0091-6749
pubmed:author
pubmed:issnType
Print
pubmed:volume
119
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1092-8
pubmed:dateRevised
2011-6-16
pubmed:meshHeading
pubmed:year
2007
pubmed:articleTitle
Role of MUC5AC in the pathogenesis of exercise-induced bronchoconstriction.
pubmed:affiliation
Department of Medicine, University of Washington, Seattle WA 98195-6522, USA. tealh@u.washington.edu
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