pubmed-article:17317842 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
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pubmed-article:17317842 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C0031727 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C0600388 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C0752312 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
pubmed-article:17317842 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
pubmed-article:17317842 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:17317842 | pubmed:dateCreated | 2007-2-23 | lld:pubmed |
pubmed-article:17317842 | pubmed:abstractText | Our studies have shown variable sensitivity of cultured melanoma cells to docetaxel. To better understand this response, we studied the role of signal transduction pathways in modulating docetaxel-induced melanoma killing. | lld:pubmed |
pubmed-article:17317842 | pubmed:language | eng | lld:pubmed |
pubmed-article:17317842 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17317842 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17317842 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17317842 | pubmed:month | Feb | lld:pubmed |
pubmed-article:17317842 | pubmed:issn | 1078-0432 | lld:pubmed |
pubmed-article:17317842 | pubmed:author | pubmed-author:HerseyPeterP | lld:pubmed |
pubmed-article:17317842 | pubmed:author | pubmed-author:ZhangXu... | lld:pubmed |
pubmed-article:17317842 | pubmed:author | pubmed-author:MhaidatNizar... | lld:pubmed |
pubmed-article:17317842 | pubmed:author | pubmed-author:JiangChen... | lld:pubmed |
pubmed-article:17317842 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17317842 | pubmed:day | 15 | lld:pubmed |
pubmed-article:17317842 | pubmed:volume | 13 | lld:pubmed |
pubmed-article:17317842 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17317842 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17317842 | pubmed:pagination | 1308-14 | lld:pubmed |
pubmed-article:17317842 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:17317842 | pubmed:meshHeading | pubmed-meshheading:17317842... | lld:pubmed |
pubmed-article:17317842 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17317842 | pubmed:articleTitle | Docetaxel-induced apoptosis of human melanoma is mediated by activation of c-Jun NH2-terminal kinase and inhibited by the mitogen-activated protein kinase extracellular signal-regulated kinase 1/2 pathway. | lld:pubmed |
pubmed-article:17317842 | pubmed:affiliation | Immunology and Oncology Unit, Royal Newcastle Hospital, Newcastle, New South Wales, Australia. | lld:pubmed |
pubmed-article:17317842 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17317842 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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