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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2007-2-5
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pubmed:abstractText |
Hepatic encephalopathy is neuropsychiatric derangement secondary to hepatic decompensation or portal-systemic shunting. Nitric oxide (NO) synthase inhibition aggravates encephalopathy and increases mortality in rats with thioacetamide (TAA)-induced acute liver failure, suggesting a protective role of NO. This study investigated the roles of endothelium-derived constitutive NO synthase (eNOS) and inducible NOS (iNOS) in the liver of rats with fulminant hepatic failure and encephalopathy.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1726-4901
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
70
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
16-23
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pubmed:meshHeading |
pubmed-meshheading:17276928-Alanine Transaminase,
pubmed-meshheading:17276928-Animals,
pubmed-meshheading:17276928-Aspartate Aminotransferases,
pubmed-meshheading:17276928-Bilirubin,
pubmed-meshheading:17276928-Blotting, Western,
pubmed-meshheading:17276928-Hepatic Encephalopathy,
pubmed-meshheading:17276928-Liver,
pubmed-meshheading:17276928-Liver Failure, Acute,
pubmed-meshheading:17276928-Male,
pubmed-meshheading:17276928-Motor Activity,
pubmed-meshheading:17276928-Nitric Oxide Synthase,
pubmed-meshheading:17276928-Rats,
pubmed-meshheading:17276928-Rats, Sprague-Dawley,
pubmed-meshheading:17276928-Thioacetamide
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pubmed:year |
2007
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pubmed:articleTitle |
Role of hepatic nitric oxide synthases in rats with thioacetamide-induced acute liver failure and encephalopathy.
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pubmed:affiliation |
Division of Gastroenterology, Department of Medicine, Taipei Veterans General Hospital, and National Yang-Ming University School of Medicine, Taipei, Taiwan, R.O.C.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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