pubmed-article:17264802 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17264802 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
pubmed-article:17264802 | lifeskim:mentions | umls-concept:C0860207 | lld:lifeskim |
pubmed-article:17264802 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:17264802 | lifeskim:mentions | umls-concept:C1412070 | lld:lifeskim |
pubmed-article:17264802 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
pubmed-article:17264802 | lifeskim:mentions | umls-concept:C0380509 | lld:lifeskim |
pubmed-article:17264802 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:17264802 | pubmed:dateCreated | 2007-1-31 | lld:pubmed |
pubmed-article:17264802 | pubmed:abstractText | Increasing evidence suggests that a genetically determined functional impairment of the hepatocellular efflux transporters bile salt export pump (BSEP, ABCB11) and multidrug resistance protein 3 (MDR3, ABCB4) play a pathophysiological role in the development of drug-induced liver injury. The aim of this study was therefore to describe the extent of genetic variability in ABCB11 and ABCB4 in patients with drug-induced liver injury and to in vitro functionally characterize newly detected ABCB11 mutations and polymorphisms. | lld:pubmed |
pubmed-article:17264802 | pubmed:language | eng | lld:pubmed |
pubmed-article:17264802 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17264802 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17264802 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17264802 | pubmed:month | Jan | lld:pubmed |
pubmed-article:17264802 | pubmed:issn | 1744-6872 | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:LangThomasT | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:MeierPeter... | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:Kullak-Ublick... | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:StiegerBrunoB | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:BeuersUlrichU | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:KerbReinholdR | lld:pubmed |
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pubmed-article:17264802 | pubmed:author | pubmed-author:LangCarmenC | lld:pubmed |
pubmed-article:17264802 | pubmed:author | pubmed-author:MeierYvonneY | lld:pubmed |
pubmed-article:17264802 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17264802 | pubmed:volume | 17 | lld:pubmed |
pubmed-article:17264802 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17264802 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17264802 | pubmed:pagination | 47-60 | lld:pubmed |
pubmed-article:17264802 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:17264802 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17264802 | pubmed:articleTitle | Mutations and polymorphisms in the bile salt export pump and the multidrug resistance protein 3 associated with drug-induced liver injury. | lld:pubmed |
pubmed-article:17264802 | pubmed:affiliation | Division of Clinical Pharmacology and Toxicology, University Hospital Zurich, Switzerland. | lld:pubmed |
pubmed-article:17264802 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17264802 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:17264802 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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