Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
2007-1-30
pubmed:abstractText
The cell-associated proteoglycan syndecan-1 (Synd1) closely regulates inflammation and cell-matrix interactions during wound healing and tumorigenesis. The present study investigated whether Synd1 may also regulate cardiac inflammation, matrix remodeling, and function after myocardial infarction (MI).
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
30
pubmed:volume
115
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
475-82
pubmed:meshHeading
pubmed-meshheading:17242279-Animals, pubmed-meshheading:17242279-Cardiomyopathy, Dilated, pubmed-meshheading:17242279-Collagen, pubmed-meshheading:17242279-Cytokines, pubmed-meshheading:17242279-Disease Models, Animal, pubmed-meshheading:17242279-Female, pubmed-meshheading:17242279-Gene Expression, pubmed-meshheading:17242279-Heart, pubmed-meshheading:17242279-Leukocytes, pubmed-meshheading:17242279-Male, pubmed-meshheading:17242279-Matrix Metalloproteinase 2, pubmed-meshheading:17242279-Matrix Metalloproteinase 9, pubmed-meshheading:17242279-Mice, pubmed-meshheading:17242279-Mice, Inbred BALB C, pubmed-meshheading:17242279-Mice, Knockout, pubmed-meshheading:17242279-Mutagenesis, pubmed-meshheading:17242279-Myocardial Infarction, pubmed-meshheading:17242279-Myocardium, pubmed-meshheading:17242279-RNA, Messenger, pubmed-meshheading:17242279-Syndecan-1, pubmed-meshheading:17242279-Systole, pubmed-meshheading:17242279-Ventricular Remodeling
pubmed:year
2007
pubmed:articleTitle
Increased expression of syndecan-1 protects against cardiac dilatation and dysfunction after myocardial infarction.
pubmed:affiliation
Experimental and Molecular Cardiology/CARIM, University Hospital Maastricht, PO Box 5800, 6202 AZ Maastricht, The Netherlands.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't