Source:http://linkedlifedata.com/resource/pubmed/id/17167474
Switch to
Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
7121
|
pubmed:dateCreated |
2006-12-14
|
pubmed:abstractText |
Metabolic and immune systems are among the most fundamental requirements for survival. Many metabolic and immune response pathways or nutrient- and pathogen-sensing systems have been evolutionarily conserved throughout species. As a result, immune response and metabolic regulation are highly integrated and the proper function of each is dependent on the other. This interface can be viewed as a central homeostatic mechanism, dysfunction of which can lead to a cluster of chronic metabolic disorders, particularly obesity, type 2 diabetes and cardiovascular disease. Collectively, these diseases constitute the greatest current threat to global human health and welfare.
|
pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:status |
MEDLINE
|
pubmed:month |
Dec
|
pubmed:issn |
1476-4687
|
pubmed:author | |
pubmed:issnType |
Electronic
|
pubmed:day |
14
|
pubmed:volume |
444
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
860-7
|
pubmed:meshHeading |
pubmed-meshheading:17167474-Adipose Tissue,
pubmed-meshheading:17167474-Animals,
pubmed-meshheading:17167474-Diabetes Mellitus, Type 2,
pubmed-meshheading:17167474-Humans,
pubmed-meshheading:17167474-Inflammation,
pubmed-meshheading:17167474-Insulin Resistance,
pubmed-meshheading:17167474-Metabolic Syndrome X,
pubmed-meshheading:17167474-Obesity
|
pubmed:year |
2006
|
pubmed:articleTitle |
Inflammation and metabolic disorders.
|
pubmed:affiliation |
Department of Genetics & Complex Diseases, Harvard School of Public Health, 665 Huntington Avenue, Boston, Massachusetts 02115, USA. ghotamis@hsph.harvard.edu
|
pubmed:publicationType |
Journal Article,
Review,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
|