pubmed-article:17166870 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17166870 | lifeskim:mentions | umls-concept:C1704837 | lld:lifeskim |
pubmed-article:17166870 | lifeskim:mentions | umls-concept:C0152171 | lld:lifeskim |
pubmed-article:17166870 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:17166870 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
pubmed-article:17166870 | lifeskim:mentions | umls-concept:C0205373 | lld:lifeskim |
pubmed-article:17166870 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:17166870 | pubmed:dateCreated | 2007-3-26 | lld:pubmed |
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pubmed-article:17166870 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17166870 | pubmed:abstractText | Systemic sclerosis (SSc) is a connective tissue disorder characterized by early generalized microangiopathy with disturbed angiogenesis. Endoglin gene (ENG) encodes a transmembrane glycoprotein which acts as an accessory receptor for the transforming growth factor-beta (TGF-beta) superfamily, and is crucial for maintaining vascular integrity. A 6-base insertion in intron 7 (6bINS) of ENG has been reported to be associated with microvascular disturbance. | lld:pubmed |
pubmed-article:17166870 | pubmed:language | eng | lld:pubmed |
pubmed-article:17166870 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17166870 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:17166870 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17166870 | pubmed:month | Apr | lld:pubmed |
pubmed-article:17166870 | pubmed:issn | 1462-0324 | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:GuillevinLL | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:JunienCC | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:MeyerOO | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:KahanAA | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:CabaneJJ | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:BoileauCC | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:HachullaEE | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:SibiliaJJ | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:MouthonLL | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:WipffJJ | lld:pubmed |
pubmed-article:17166870 | pubmed:author | pubmed-author:AllanoreYY | lld:pubmed |
pubmed-article:17166870 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17166870 | pubmed:volume | 46 | lld:pubmed |
pubmed-article:17166870 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17166870 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17166870 | pubmed:pagination | 622-5 | lld:pubmed |
pubmed-article:17166870 | pubmed:dateRevised | 2007-9-6 | lld:pubmed |
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pubmed-article:17166870 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17166870 | pubmed:articleTitle | Association between an endoglin gene polymorphism and systemic sclerosis-related pulmonary arterial hypertension. | lld:pubmed |
pubmed-article:17166870 | pubmed:affiliation | René Descartes University, Medical Faculty, Rheumatology A Department, Cochin Hospital, AP-HP, Paris, France. | lld:pubmed |
pubmed-article:17166870 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17166870 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:17166870 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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