Source:http://linkedlifedata.com/resource/pubmed/id/17164797
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2007-2-22
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pubmed:abstractText |
With the availability of dense maps of anonymous and frequent SNPs spanning the whole human genome, genome-wide association studies are now becoming a reality. In this paper, we discuss the utility of these approaches to detect genetic risk variants involved in complex disease susceptibility and, in the best case scenario where a signal is detected, how helpful it will be to the understanding of the pathological process.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
1018-4813
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
15
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
260-3
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pubmed:meshHeading | |
pubmed:year |
2007
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pubmed:articleTitle |
Are genome-wide association studies all that we need to dissect the genetic component of complex human diseases?
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pubmed:affiliation |
1INSERM U535, University Paris Sud, Villejuif F-94817, France. bourgain@vjf.inserm.fr
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pubmed:publicationType |
Journal Article
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